| Type | MGI:General | Description | following electrical stimulation, mutant EDL muscles exhibit increased nitric oxide (NO) metabolism (a 2.1-fold increase in cGMP formation) relative to wild-type EDL muscles in mutant EDS muscles, this increase is presumed to promote oxygen delivery by a NO-mediated vasodilation mechanism and is shown to be attenuated by a nitric oxide synthase inhibitor no significant increase in cGMP is observed between stimulated wild-type and mutant soleus muscles |
