Type | Indexed | Publication . Mgi Jnum | J:269422 |
Publication . Citation | Hilton JB, et al. (2018) The accumulation of enzymatically inactive cuproenzymes is a CNS-specific phenomenon of the SOD1(G37R) mouse model of ALS and can be restored by overexpressing the human copper transporter hCTR1. Exp Neurol 307:118-128 |