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Protein Coding Gene : Atp5pf ATP synthase peripheral stalk subunit F6

Primary Identifier  MGI:107777 Organism  mouse, laboratory
Chromosome  16 NCBI Gene Number  11957
Mgi Type  protein coding gene
description  FUNCTION: Automated description from the Alliance of Genome Resources (Release 7.0.0)

Predicted to enable proton transmembrane transporter activity. Predicted to contribute to ATP hydrolysis activity and proton-transporting ATP synthase activity, rotational mechanism. Predicted to be involved in several processes, including negative regulation of icosanoid secretion; positive regulation of heart rate; and proton motive force-driven mitochondrial ATP synthesis. Predicted to act upstream of or within proton transmembrane transport. Located in mitochondrial inner membrane. Is expressed in several structures, including alimentary system; central nervous system; genitourinary system; respiratory system; and sensory organ. Orthologous to human ATP5PF (ATP synthase peripheral stalk subunit F6).
  • synonyms:
  • ATP synthase, H+ transporting. mitochondrial F0 complex subunit F,
  • ATP synthase, H+ transporting, mitochondrial F0 complex, subunit F,
  • MGD-MRK-36354,
  • ATP synthase peripheral stalk subunit F6,
  • Atp5j,
  • Atp5pf

Features --> Cross References

Genome

Sequence Feature Displayer

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0 Canonical

0 CDSs

0 Exons

0 Genomic Clusters

4 Involved In Mutations

0 Strain

0 Transcripts

0 Transgenic Expressors

0 UTRs

Canonical gene --> CDSs in specific strains.

Canonical gene --> Exons in specific strains

Canonical gene --> Strain-specific IDs, biotypes, and locations

Canonical gene --> Transcripts in specific strains.

Features --> Overlapping features

Proteins

Gene --> Proteins

Function

Mouse features --> Functions (GO terms)

Homology

Genes --> Homologs

Interactions

9 Pathways

0 Targeted By

Gene --> Protein-Protein Interactions

Expression

Gene --> Expression annotations

Phenotype

Genes/Features --> Phenotypes (MP terms)

Disease

Mouse features --> Human diseases

Literature

Mouse features --> Publications

 

Other

0 Driver For