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Protein Coding Gene : Ap1ar adaptor-related protein complex 1 associated regulatory protein

Primary Identifier  MGI:2384822 Organism  mouse, laboratory
Chromosome  3 NCBI Gene Number  211556
Mgi Type  protein coding gene
description  FUNCTION: Automated description from the Alliance of Genome Resources (Release 7.1.0)

Predicted to enable AP-1 adaptor complex binding activity; Arp2/3 complex binding activity; and kinesin binding activity. Acts upstream of or within several processes, including negative regulation of cell motility; negative regulation of substrate adhesion-dependent cell spreading; and regulation of Arp2/3 complex-mediated actin nucleation. Located in endosome. Is expressed in several structures, including cardiovascular system; genitourinary system; gut; integumental system; and nervous system. Orthologous to human AP1AR (adaptor related protein complex 1 associated regulatory protein).
  • synonyms:
  • expressed sequence C78909,
  • AA407621,
  • Ap1ar,
  • Gadkin,
  • C78909,
  • cDNA sequence BC002199,
  • expressed sequence AA407621,
  • BC002199,
  • MGI:2140060,
  • MGI:2139553,
  • adaptor-related protein complex 1 associated regulatory protein

Features --> Cross References

Genome

Sequence Feature Displayer

JG Browse Displayer

0 Canonical

0 CDSs

0 Exons

0 Genomic Clusters

2 Involved In Mutations

0 Strain

0 Transcripts

0 Transgenic Expressors

0 UTRs

Canonical gene --> CDSs in specific strains.

Canonical gene --> Exons in specific strains

Canonical gene --> Strain-specific IDs, biotypes, and locations

Canonical gene --> Transcripts in specific strains.

Features --> Overlapping features

Proteins

Gene --> Proteins

Function

Mouse features --> Functions (GO terms)

Homology

Genes --> Homologs

Interactions

0 Pathways

0 Targeted By

Gene --> Protein-Protein Interactions

Expression

Gene --> Expression annotations

Phenotype

Genes/Features --> Phenotypes (MP terms)

Disease

Mouse features --> Human diseases

Literature

Mouse features --> Publications

 

Other

0 Driver For