| First Author | Yang G | Year | 2009 |
| Journal | Br J Cancer | Volume | 100 |
| Issue | 12 | Pages | 1912-6 |
| PubMed ID | 19455142 | Mgi Jnum | J:151099 |
| Mgi Id | MGI:4352781 | Doi | 10.1038/sj.bjc.6605087 |
| Citation | Yang G, et al. (2009) Mitochondrial dysfunction resulting from loss of cytochrome c impairs radiation-induced bystander effect. Br J Cancer 100(12):1912-6 |
| abstractText | Cytochrome c is a pivotal protein that resides in mitochondria as component of mitochondria respiration and apoptosis initiator. Using murine cells lacking cytochrome c, we showed here that cytochrome c-deficient cells had attenuated reactive oxygen species/nitric oxide and micronuclei induction to radiation-induced bystander signals, indicating cytochrome c is essential for the bystander effect. |
